association of the apolipoprotein a5 gene ?1131tc polymorphism with serum lipids in korean subjects impact of sasang constitution载脂蛋白a5基因协会 1131 tc多态性与血清脂质在韩国sasang宪法主体的影响.pdfVIP

association of the apolipoprotein a5 gene ?1131tc polymorphism with serum lipids in korean subjects impact of sasang constitution载脂蛋白a5基因协会 1131 tc多态性与血清脂质在韩国sasang宪法主体的影响.pdf

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association of the apolipoprotein a5 gene ?1131tc polymorphism with serum lipids in korean subjects impact of sasang constitution载脂蛋白a5基因协会 1131 tc多态性与血清脂质在韩国sasang宪法主体的影响

Hindawi Publishing Corporation Evidence-Based Complementary and Alternative Medicine Volume 2012, Article ID 598394, 8 pages doi:10.1155/2012/598394 Research Article Association of the Apolipoprotein A5 Gene −1131TC Polymorphism with Serum Lipids in Korean Subjects: Impact of Sasang Constitution Kwang Hoon Song, Sung-Gon Yu, Seongwon Cha, and Jong Yeol Kim Division of Constitutional Medicine Research, Korea Institute of Oriental Medicine, 483 Exporo, Yuseong-gu, Daejeon 305811, Republic of Korea Correspondence should be addressed to Jong Yeol Kim, ssmed@kiom.re.kr Received 24 February 2011; Revised 19 April 2011; Accepted 17 May 2011 Academic Editor: Jenny M. Wilkinson Copyright © 2012 Kwang Hoon Song et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Apolipoprotein A5 (APOA5 ) was identified as a strong modulator of serum lipids. Moreover, an APOA5 gene −1131TC polymorphism has been associated with serum lipids, but the results are inconsistent according to ethnic and racial groups. We have genotyped and analyzed 1,619 outpatients of Korean oriental medicine hospitals who were classified into three Sasang constitution groups (SCGs), So-Yang (SY), So-Eum (SE), and Tae-Eum (TE). There were no significant difference in the distribution of the APOA5 −1131TC genotype among the three SCGs. Subjects with the C allele in SY and TE showed significantly lower serum high-density lipoprotein cholesterol (HDL-C) and higher triglyceride (TG) levels than noncarriers of the C allele. These results show the differences in the prevalence of decreasing serum HDL-C and elevating serum TG l

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