原创力文档regulation of gabaa and glutamate receptor expression, synaptic facilitation and long-term potentiation in the hippocampus of prion mutant micegabaa和谷氨酸受体表达,调节突触便利化和长期势差朊病毒突变小鼠的海马.pdfVIP
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regulation of gabaa and glutamate receptor expression, synaptic facilitation and long-term potentiation in the hippocampus of prion mutant micegabaa和谷氨酸受体表达,调节突触便利化和长期势差朊病毒突变小鼠的海马
Regulation of GABAA and Glutamate Receptor
Expression, Synaptic Facilitation and Long-Term
Potentiation in the Hippocampus of Prion Mutant Mice
1,2 ˜ 3 ` ´ 3 ´ 1,2 1,2
Alejandra Rangel , Noelia Madronal , Agnes Gruart i Masso , Rosalina Gavın , Franc Llorens , Lauro
4 ´ 5 ´ ´ ´ 3 ´ ´ 1,2
Sumoy , Juan Marıa Torres , Jose Marıa Delgado-Garcıa *, Jose Antonio Del Rıo *
1 Molecular and Cellular Neurobiotechnology, Institute for Bioengineering of Catalonia, and Department of Cell Biology, University of Barcelona, Barcelona, Spain,
´ ´
2 Centro de Investigacion Biomedica en Red de Enfermedades Neurodegenerativas (CIBERNED), Madrid, Spain, 3 Division de Neurociencias. Universidad Pablo de Olavide,
´
Sevilla, Spain, 4 Institute of Predictive and Personalized Medicine of Cancer, Badalona, Spain, 5 Centro de Investigacion en Sanidad Animal (CISA), INIA, Valdeolmos,
Madrid, Spain
Abstract
Background: Prionopathies are characterized by spongiform brain degeneration, myoclonia, dementia, and periodic
electroencephalographic (EEG) disturbances. The hallmark of prioniopathies is the presence of an abnormal conformational
isoform (PrPsc c
) of the natural cellular prion protein (PrP ) encoded by the Prnp gene. Although sever
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