Saccharomyces cerevisiae FKBP12 binds Arabidopsis thaliana TOR and its expression in plants leads to rapamycin susceptibility 英文参考文献.docVIP

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Saccharomyces cerevisiae FKBP12 binds Arabidopsis thaliana TOR and its expression in plants leads to rapamycin susceptibility 英文参考文献.doc

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Saccharomyces cerevisiae FKBP12 binds Arabidopsis thaliana TOR and its expression in plants leads to rapamycin susceptibility 英文参考文献

BMC Plant Biology BioMedCentral Research article Open Access Saccharomyces cerevisiae FKBP12 binds Arabidopsis thaliana TOR and its expression in plants leads to rapamycin susceptibility Rodnay Sormani1, Lei Yao1,2, Beno?t Menand3, Najla Ennar1, Cécile Lecampion1, Christian Meyer4 and Christophe Robaglia*1 Address: 1DSV-DEVM Laboratoire de Génétique et de Biophysique des Plantes, UMR 6191 CNRS-CEA-Université de la Méditerranée, Faculté des Sciences de Luminy,163 Avenue de Luminy, 13009 Marseille France, 2Beijing Agro-Biotechnology Research Center, Beijing Academy of Agriculture and Forestry Sciences. P.O. Box 2449, 100097 Beijing, China, 3Cell Developmental Biology Department, John Innes Centre, Norwich Research Park, Colney, Norwich, Norfolk, NR4 7UH, UK and 4Unité de Nutrition Azotée des Plantes, Institut Jean-Pierre Bourgin (IJPB) INRA 78026 VERSAILLES Cedex, France Email: Rodnay Sormani - Rodnay.Sormani@versailles.inra.fr; Lei Yao - yaolei@; Beno?t Menand - benoit.menand@bbsrc.ac.uk; Najla Ennar - ennar@luminy.univ-mrs.fr; Cécile Lecampion - lecampion@luminy.univ-mrs.fr; Christian Meyer - meyer@versailles.inra.fr; Christophe Robaglia* - robaglia@luminy.univ-mrs.fr * Corresponding author Published: 1 June 2007 Received: 13 December 2006 Accepted: 1 June 2007 BMC Plant Biology 2007, 7:26 doi:10.1186/1471-2229-7-26 This article is available from: /1471-2229/7/26 ? 2007 Sormani et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Abstract Background: The eukaryotic TOR pathway controls translation, growth and the cell cycle in response to environmental signals such as nutrients or growth-stimulating factors. The TOR protein kinase can be inactivated by the antibiotic rapamycin following the formation