shock休克英文专题知识讲座.ppt

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Chapter11Shock;What’sshock?;hypotension?;vasoconstrictivetreatment→vasodilativetreatment;Lartin

GeneralCriticalReaction:strikeandvibration

1731,HenriFrancoisLeDran

applytomedicineforthefirsttime;paleface

cyanosis

coldandclammylimbs

oliguria

faintconsciousness

hypotension;“sympatheticfailuretheory”;bloodvolume↓

ECBV(effectivecirculatingbloodvolume)↓→microcirculationdisturbances

sympatheticactivation;systemicinflammatoryresponsesyndrome,SIRS全身炎症反应综合征

pro-inflammatoryanti-inflammatoryfactors;Regardlessofcauses,shockisdefinedasaclinicalconditionandpathophysiologicprocesscharacterizedbymicrocirculationdysfunction(inadequatebloodflowvitalorgans,andinabilityofcelltometabolizenormally).

causes→acutecirculatoryfailure→microcirculationdisturbance→ECBV(effectivecirculatorybloodvolume)↓→“lowflowstate”invitalorgans→abnormalcellularmetabolismandfunction,tissueinjury,dysfunctionofvitalorgans→shock;1.Lossofbloodorfluid:

Bloodloss:pepticulcer,varicesduetoportalhypertension

Fluidloss:severevomiting,diarrhea,intestinalobstruction

(collapse)

2.Burn:

earlystage:lossofbodyfluid,pain

laterstage:lossofbodyfluid,infection

3.Trauma:

hemorrhage,severepain

4.Infection:

G-infection→releaseofendotoxins

sepsis;5.Anaphylaxis:;6.Strongstimulationonnervesystem:

braininjury,thedepressantactionofdrugs,generalanesthesia,hypoxia,orlackofglucose,spinalanesthesiaorspinalcordinjury→decreasedsympatheticcontrol;AccordingtoEtiology:;

bloodvolume

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