JNK2fSAPK、PPARγ、eIF4E在食管癌变过程中的表达及其意义.pdfVIP

英文摘要 further the ofESCC．To explore carcinogenesis of carcinoma，theof esophageal expression carcinogenesis eIF4Einthe normal 肿己P—JNK，PPAR?andesophageal and cellcarcinoma mucosa，atypicalhyperplasiasquamous were studiedwith comparatively staining． Method：1The of expression elF4Ewasstudiedin15 casegofnormal esophageal casesof 24casesof epithelium；70dysplasia(including casesof moderate—grade low-grade dysplasia, dysplasia，26 20casesof casesof hiI曲一gradedysplasia)；75esophageal in carcinoma cell 15casesof carcinoma(including squamous cell casesofwell．differentiated carcinoma， situ，20 squamous 20casesof differentiated cell moderately squamous and20casesof differentiated carcinoma poorly squamous cell Power carcinoma)by method．Theclinical immunohistochemical staining of